Frontiers Lysosomal and Mitochondrial Liaisons in Niemann-Pick

Mitochondrial biogenesis is transcriptionally repressed in lysosomal lipid storage diseases

A proposed model for the pathogenesis of Niemann-Pick disease type

Intracellular Cholesterol Trafficking and Impact in Neurodegeneration. - Abstract - Europe PMC

Correction of Niemann-Pick type C1 trafficking and activity with the histone deacetylase inhibitor valproic acid - ScienceDirect

Frontiers Lysosomes and Cancer Progression: A Malignant Liaison

PDF) Lysosomal and Mitochondrial Liaisons in Niemann-Pick Disease

Massive Accumulation of Sphingomyelin Affects the Lysosomal and Mitochondria Compartments and Promotes Apoptosis in Niemann-Pick Disease Type A

Niemann-Pick type C2 deficiency impairs autophagy-lysosomal activity, mitochondrial function, and TLR signaling in adipocytes - ScienceDirect

Correction of Niemann-Pick type C1 trafficking and activity with the histone deacetylase inhibitor valproic acid - ScienceDirect

Mitochondrial biogenesis is transcriptionally repressed in lysosomal lipid storage diseases

PDF) Acid ceramidase improves mitochondrial function and oxidative stress in Niemann-Pick type C disease by repressing STARD1 expression and mitochondrial cholesterol accumulation

IJMS, Free Full-Text

Niemann–Pick type C disease: cellular pathology and pharmacotherapy - Wheeler - 2020 - Journal of Neurochemistry - Wiley Online Library

Niemann-Pick type C2 deficiency impairs autophagy-lysosomal activity, mitochondrial function, and TLR signaling in adipocytes - ScienceDirect