The GluN1, GluN2A, and GluN2B pre-M1 linker is intolerant to genetic
Por um escritor misterioso
Last updated 01 abril 2025


Opportunities for Precision Treatment of GRIN2A and GRIN2B Gain-of
Molecular Mechanism of Disease-Associated Mutations in the Pre-M1

Positive allosteric modulators that target NMDA receptors rectify

Frontiers Imprecision in Precision Medicine: Differential

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De Novo Mutations and Rare Variants Occurring in NMDA Receptors

Structural Basis of the Proton Sensitivity of Human GluN1-GluN2A

The GluN1, GluN2A, and GluN2B pre-M1 linker is intolerant to

Frontiers Protein quality control of N-methyl-D-aspartate receptors

Cross-subunit interactions that stabilize open states mediate

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Intolerance analysis of genetic variation across functional

Cross-subunit interactions that stabilize open states mediate
Molecular Mechanism of Disease-Associated Mutations in the Pre-M1

The role of NMDA receptor and neuroligin rare variants in synaptic
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